Concussions May Damage Areas Of The Brain Related To Memory

Concussions May Damage Areas Of The Brain Related To Memory.
Concussions may devastation areas of the sense related to memory in National Football League players. And that expense might linger long after the players leave the sport, according to a small study. "We're hoping that our findings are prospering to further inform the game," Dr Jennifer Coughlin, an deputy professor of psychiatry and behavioral sciences at Johns Hopkins University School of Medicine in Baltimore, said in a university front-page news release recommended reading. "That may mean individuals are able to make more educated decisions about whether they're gullible to brain injury, advise how helmets are structured or inform guidelines for the adventurous enough to better protect players".

Hypothyroidism Affects The Brain

Hypothyroidism Affects The Brain.
Hypothyroidism, a teach that causes low or no thyroid hormone production, is not linked to unassuming dementia or impaired brain function, a new review suggests. Although more research is needed, the scientists said their findings add to mounting validation that the thyroid gland disorder is not tied to the memory and thinking problems known as "mild cognitive impairment" bestvito.men. Some previous evidence has suggested that changes in the body's endocrine system, including thyroid function, might be linked to Alzheimer's illness and other forms of dementia, said researchers led by Dr Ajay Parsaik, of the University of Texas Medical School in Houston.

Mild cognitive impairment, in particular, is intellect to be an antiquated warning sign of the memory-robbing disorder Alzheimer's disease, the contemplation authors said in a university news release. In conducting the study, Parsaik's rig examined a group of more than 1900 people, including those with mild and more severe cases of hypothyroidism as example. The participants, who were from the same Minnesota county, were between 70 and 89 years of age.

High Blood Pressure May Prognosticate Dementia in Some Elderly Peoples

High Blood Pressure May Prognosticate Dementia in Some Elderly Peoples.
High blood weight may announce dementia in older adults with impaired executive responsibility (difficulty organizing thoughts and making decisions), but not in those with memory problems, a new study has found herbalm.top. The observe included 990 dementia-free participants, average age 83, who were followed-up for five years.

During that time, dementia developed in 59,5 percent of those with and in 64,2 percent of those without tall blood pressure extenderdeluxe.com. Similar rates were seen in participants with remembrance dysfunction alone and with both memory and chief executive dysfunction.

However, among those with executive dysfunction alone, the rate of dementia development was 57,7 percent in the midst those with high blood pressure compared to 28 percent for those without high blood pressure, which is also called hypertension. "We show herein that the association of hypertension predicts progression to dementia in a subgroup of about one-third of subjects with cognitive impairment, no dementia," wrote the researchers at the University of Western Ontario in Canada.

So "Control of hypertension in this populace could contract by one-half the projected 50-percent five-year rate of movement forward to dementia." The study findings are published in the February issue of the journal Archives of Neurology. The findings may affirm important for elderly people with cognitive impairment but no dementia, the investigation authors noted.

Many Women In The First Year After Menopause Deteriorating Memory And Fine Motor Skills

Many Women In The First Year After Menopause Deteriorating Memory And Fine Motor Skills.
Women present through menopause occasionally deem they are off their mental game, forgetting phone numbers and passwords, or struggling to find a particular word. It can be frustrating, discomfiting and worrisome, but a small new study helps to explain the struggle. Researchers found that women in the foremost year after menopause perform slightly worse on certain loony tests than do those who are approaching their post-reproductive years. "This study shows, as have others, that there are cognitive conceptual declines that are real, statistically significant and clinically significant," said study author Miriam Weber, an subsidiary professor in the department of neurology at the University of Rochester in Rochester, NY "These are tricky declines in performance, so women aren't becoming globally impaired and unable to function extreme. But you criticism it on a daily basis".

The study is published in the current issue of the journal Menopause. According to the researchers, the ready of learning, retaining and applying new information is associated with regions of the knowledge that are rich in estrogen receptors. The natural fluctuation of the hormone estrogen during menopause seems to be linked to problems associated with thoughtful and memory. "We found the problem is not related to absolute hormone levels pro extender manual berlin. Estrogen declines in the transition, but before it falls, there are effective fluctuations".

Weber explained that it is the variation in estrogen knock down that most likely plays a critical role in creating the memory problems many women experience. As the body readjusts to the changes in hormonal levels any time after a woman's period stops, the researchers think mental challenges diminish. While Weber said it is important that women get wind that memory issues associated with menopause are most likely normal and temporary, the study did not include women whose periods had stopped for longer than one year. Weber added that she plans to pinpoint more just how long-term honour and thinking problems persist in a future study.

Other research has offered conflicting conclusions about the cerebral changes associated with menopause, the study authors wrote. The Chicago situate of the Study of Women's Health Across the Nation (SWAN) initially found no relation between what stage of menopause women were in and how they performed on tests of working recollection or perceptual speed. However, a different SWAN examine identified deficits in memory and processing speed in the late menopausal stage.

Studies of menopause typically circumscribe distinct stages of menopause, although researchers may differ in where they draw the line between those transitions. The researchers twisted with this study said that the variation in findings between studies may be due to different ways of staging menopause.

Doctors Discovered How The Brain Dies

Doctors Discovered How The Brain Dies.
Shrunken structures also gaol the brains of profound marijuana users might explain the stereotype of the "pothead," brain researchers report. Northwestern University scientists studying teens who were marijuana smokers or departed smokers found that parts of the thought related to working memory appeared diminished in size - changes that coincided with the teens' poor as a church-mouse performance on memory tasks free neosize xl sample. "We observed that the shapes of brain structures interrelated to short-term memory seemed to collapse inward or shrink in people who had a history of routine marijuana use when compared to healthy participants," said study author Matthew Smith.

He is an auxiliary research professor in psychiatry and behavioral sciences at the Northwestern University Feinberg School of Medicine, in Chicago. The shrinking of these structures appeared to be more advanced in common people who had started using marijuana at a younger age. This suggests that youngsters might be more vulnerable to drug-related memory loss, according to the study, which was published in the Dec 16 bataye. 2013 descendant of the journal Schizophrenia Bulletin.

So "The brain abnormalities we're observing are entirely related to poor short-term memory performance. The more that sagacity looks abnormal, the poorer they're doing on memory tests". The paper is provocative because the participants had not been using marijuana for a unite years, indicating that memory problems might persist even if the person quits smoking the drug, said Dr Frances Levin, chairman of the American Psychiatric Association's Council on Addiction Psychiatry. At the same time, Levin cautioned that the also scratch paper presents a chicken-or-egg problem.

It's not understandably whether marijuana use caused the thought problems or people with memory problems tended to use marijuana. "The big $64000 mystery is whether these memory problems predate the marijuana use". The examine focused on nearly 100 participants sorted into four groups: healthy people who never used pot, strong people who were former heavy pot smokers, people with schizophrenia who never used paunch and schizophrenics who were former heavy pot users. Researchers used MRI scans to den the structure of participants' brains.

New Solutions For The Prevention Of Memory Loss From Multiple Sclerosis

New Solutions For The Prevention Of Memory Loss From Multiple Sclerosis.
Being mentally acting may helper reduce memory and learning problems that often arise in people with multiple sclerosis, a new study suggests. It included 44 people, about period 45, who'd had MS for an average of 11 years. Even if they had higher levels of imagination damage, those with a mentally active lifestyle had better scores on tests of learning and recollection than those with less intellectually enriching lifestyles neosize xl shop. "Many people with MS struggle with learning and memory problems," examination author James Sumowski, of the Kessler Foundation Research Center in West Orange, NJ, said in an American Academy of Neurology tidings release.

So "This study shows that a mentally powerful lifestyle might reduce the harmful effects of brain damage on learning and memory. Learning and thought ability remained quite good in people with enriching lifestyles, even if they had a lot of wit damage brain atrophy as shown on brain scans ," Sumowski continued khilakar. "In contrast, persons with lesser mentally running lifestyles were more likely to suffer learning and memory problems, even at milder levels of perceptiveness damage".

Sumowski said the "findings suggest that enriching activities may build a person's 'cognitive reserve,' which can be regard of as a buffer against disease-related memory impairment. Differences in cognitive retain among persons with MS may explain why some persons suffer memory problems early in the disease, while others do not expand memory problems until much later, if at all".

The study appears in the June 15 spring of Neurology. In an editorial accompanying the study, Peter Arnett of Penn State University wrote that "more digging is needed before any firm recommendations can be made," but that it seemed right to encourage people with MS to get involved with mentally challenging activities that might improve their cognitive reserve.

What is Multiple Sclerosis? An unpredictable disability of the central nervous system, multiple sclerosis (MS) can organize from relatively benign to somewhat disabling to devastating, as communication between the brain and other parts of the body is disrupted. Many investigators into MS to be an autoimmune disease - one in which the body, through its invulnerable system, launches a defensive attack against its own tissues. In the case of MS, it is the nerve-insulating myelin that comes under assault. Such assaults may be linked to an unresearched environmental trigger, it may be a virus.

Most people experience their first symptoms of MS between the ages of 20 and 40; the prime symptom of MS is often blurred or double vision, red-green color distortion, or even blindness in one eye. Most MS patients involvement muscle weakness in their extremities and difficulty with coordination and balance. These symptoms may be unbending enough to impair walking or even standing. In the worst cases, MS can manufacture partial or complete paralysis.

In A Study Of The Alzheimer'S Disease There Is A New Discovery

In A Study Of The Alzheimer'S Disease There Is A New Discovery.
New scrutinize could alter the way scientists view the causes - and budding prevention and treatment - of Alzheimer's disease. A study published online this month in the Annals of Neurology suggests that "floating" clumps of amyloid beta (abeta) proteins called oligomers could be a brief cause of the disorder, and that the better-known and more stationary amyloid-beta plaques are only a ex- mark of the disease vimaxpill.men. "Based on these and other studies, I think that one could now fairly revise the 'amyloid hypothesis' to the 'abeta oligomer hypothesis,'" said leadership researcher Dr Sam Gandy, a professor of neurology and psychiatry and associate director of the Alzheimer's Disease Research Center at Mount Sinai School of Medicine in New York City.

The novel study could herald a major swerve in Alzheimer's research, another expert said. Maria Carrillo, senior director of medical and painstaking relations at the Alzheimer's Association, said that "we are excited about the paper. We think it has some very gripping results and has potential for moving us in another direction for future research" bestvito. According to the Alzheimer's Association, more than 5,3 million Americans now be reduced from the neurodegenerative illness, and it is the seventh leading cause of death.

There is no effective curing for Alzheimer's, and its origins remain unknown. For decades, research has focused on a buildup of amyloid beta plaques in the brain, but whether these deposits are a cause of the plague or merely a neutral artifact has remained unclear. The brand-new study looked at a lesser-known factor, the more mobile abeta oligomers that can serve as in brain tissue.

In their research, Gandy's team first developed mice that only form abeta oligomers in their brains, and not amyloid plaques. Based on the results of tests gauging spatial wisdom and memory, these mice were found to be impaired by Alzheimer's-like symptoms. Next the researchers inserted a gene that would cause the mice to expose both oligomers and plaques.

Similar to the oligomer-only rodents, these mice "were still homage impaired, but no more thought impaired for having plaques superimposed on their oligomers". Another result further strengthened the notion that oligomers were the peak cause of Alzheimer's in the mice. "We tested the mice and they lost memory function, and when they died, we majestic the oligomers in their brains. Lo and behold, the degree of memory loss was proportional to the oligomer level".